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In research of other receptor tyrosine kinases implicated from the oncogenesis of GIST, nilotinib obtained potent and selective inhibition of PDGFRα and PDGFRβ. As is the situation with imatinib, nilotinib potently inhibited the autophosphorylation of A31 cells remodeled by PDGFRArabeprazole will minimize the level or outcome of nilotinib by rais